omplex information, making them “smart” devices that are
more responsive to changing patient needs.
Pacemaker Response
There are two basic ways in which pacemakers can initiate
impulses:
Triggered: These are fixed-rate pacemakers that fire
according to a predetermined plan, regardless of
the patient’s underlying cardiac activity.
Inhibited: These pacemakers are demand pacemakers—
they fire only when needed. They are capable
of inhibiting their stimulus when they sense a
patient’s complex.
It is possible for pacemakers to be both triggered and
inhibited. That is, they ignore atrial complexes but hold back
if they sense a ventricular beat.
Electrode Placement for
Temporary Pacemaker
When a patient is unstable and the EKG indicates that an
artificial pacemaker is needed, temporary pacing can be
initiated. The most common electrode placement for temporary pacemaker placement in acute settings is via external
pacing pads. Both pads can be placed on anterior chest wall,
or one pad can go on the front chest and the other on the
back. The key point when positioning the electrodes is to
ensure that the heart is well situated between the two pads
(Figure E2).
Temporary pacing can also be achieved transvenously
by inserting a special pacing wire cannula through a large
vein like the internal jugular or subclavian. Generally, this is
done only in settings where a sterile field can be maintained
and imaging is available to guide wire placement.
EKG Analysis
The electrical impulses produced by the pacemaker appear on
the EKG tracing as unnaturally sharp spikes superimposed
on the patient’s underlying rhythm. Pacemaker spikes can
be small and difficult to detect, so EKG machines often
augment the signal to make spikes more visible.
When the pacemaker captures, it produces an EKG
wave consistent with the chamber being paced. That is, if
it’s an atrial pacemaker, the spike will be followed immediately by a P wave, but if the pacemaker is stimulating the
ventricles, the spike will be followed immediately by a wide
QRS similar to a PVC. If both chambers are paced, there will
be two spikes for each cardiac cycle. See Figures E3–E5.
Properly Functioning
Pacemakers
In Figures E3–E7, the sharp spikes produced by pacemakers
are marked with a P .
Figure E2 Pacemaker Placement
TEMPORARY
PACEMAKERS
Transvenous PERMANENT PACEMAKERS
FRONT BACK
Transcutaneous
Pacemakers 537
Figure E3 Paced Complexes
A B F
P P C D E P G
Pacemakers often supplement, rather than supplant, the patient’s own rhythm. This rhythm strip shows a ventricular
demand pacemaker that fires occasionally to supplement the patient’s underlying atrial fibrillation.
The narrow complexes (C, D, E, and G) are the patient’s normally conducted beats.
The wider complexes (A, B, and F) are pacemaker-initiated (note spikes preceding each QRS).
Figure E4 Pacemaker Rhythms
P P P P P P
P
P
P
P
P
P
P
P
P
P
P
P
P
P
(a)
(b)
A properly functioning pacemaker will show a sharp spike, followed immediately by depolarization of the chamber it is
intended to pace. (a) Ventricular Pacemaker: This rhythm strip shows ventricular pacemaker capturing every beat. Note
absence of mappable P waves and sharp spikes followed immediately by wide QRS complexes. (b) AV Synchronous
Pacemaker: This rhythm strip shows an AV synchronous pacemaker capturing every beat. Note pacemaker spikes
preceding both atrial and ventricular depolarization.
538 Appendix E
Figure E5 Properly Functioning Ventricular Demand Pacemaker
P P P P
This pacemaker is set to fire whenever the patient’s intrinsic rate falls below 60 bpm. The first three complexes show
the patient’s heart depolarizing at about 68 bpm, so the pacemaker inhibited itself. It kicked in at the fourth complex
when the rate dropped and continued to pace at a rate of 60 bpm.
Figure E6 Failure to Capture/Failure to Sense
P P P P P
The underlying rhythm is atrial fibrillation, and the ventricular pacemaker is set at an unusually slow rate of 44 bpm. It
fails to sense the patient’s own complexes and continues to fire regularly, regardless of underlying ventricular activity.
When the pacemaker stimulus finds the ventricles refractory, it fails to capture.
Pacemaker Malfunction
Today’s pacemakers are generally reliable, but they still
experience occasional problems. Some of the more common
malfunctions are listed below.
Failure to Capture: Occasionally, a pacemaker fires
normally but no capture occurs; this is called a
non-capturing pacemaker. This is seen on the
EKG as pacemaker spikes that are not followed
by depolarization complexes.
Competition: If a demand pacemaker fails to sense
an underlying rhythm, it can fire in complete
disregard to the patient’s own rhythm, thereby
competing for control of the heart. Competition
can also occur if the heart’s intrinsic pacemaker
fires in a patient with a fixed-rate pacemaker, thus
allowing competition between the two pacemakers.
In either case, there is danger that the pacemaker
spike can fall during the heart’s relative refractory
period (downslope of the T wave) and instigate a
rapid, repetitive rhythm such as VT or VF.
Less common pacemaker problems include:
Runaway Pacemaker: This occurs when the pacemaker
fires too rapidly but still captures, causing the heart
to respond in a tachycardia. The problem is usually
caused by a malfunctioning impulse generator.
Battery Failure: As with all battery-operated products,
the pacemaker is useless when the battery fails.
This would appear on the EKG as the complete
absence of pacemaker spikes in a rhythm that
would normally be paced.
Pacemakers 539
Figure E7 Competition
P P P P P P P P P P
This strip shows pacemaker spikes at a rate of 100 bpm, with an underlying ventricular tachycardia/fibrillation. The
pacemaker is competing for control of the heart, but the irritable ventricular foci are winning.
Patient Assessment
Assessment must begin with the patient. Check pulses and
other perfusion parameters. Remember that the EKG can’t
tell you whether or not the heart is actually pumping blood
in response to the pacemaker’s stimuli.
Next, determine whether the pacemaker is doing what it
was intended to do. By establishing the relationship between
pacemaker spikes and the patient’s complexes, you should
be able to answer the following questions:
1. Does each spike capture?
2. Is the rate reasonable?
3. Are spikes competing with the patient’s rhythm?
4. Are spikes falling near T waves?
5. Is the pacemaker functioning consistently and reliably?
Patient Management
Pacemakers are themselves a treatment; hence, it is a mistake
to try to “treat” a malfunctioning pacemaker. When pacemakers malfunction, treatment is directed at the patient and
the underlying rhythm. See Figures E8–E19.
Sample Pacemaker Rhythm Strips
Figure E8 Fully Functional Atrial Pacemaker Showing 100% Capture
540 Appendix E
Figure E9 Ventricular Pacemaker Showing 100% Capture (with Underlying Complete Heart Block)
Figure E10 AV Sequential (DVI) Pacemaker
Figure E11 Properly Functioning Demand Pacemaker
Pacemakers 541
Figure E12 DDD Pacemaker in AV Sequential Mode
Figure E13 Sinus Rate Accelerates and Regains Control from Ventricular Demand Pacemaker
Figure E14 Pacemaker Fails to Depolarize Consistently, Indicating Lead Fracture or Displacement
542 Appendix E
Figure E15 Ventricular Pacemaker (with Underlying Complete Heart Block) with 100% Capture
Figure E16 Atrial Pacemaker with 100% Capture
Figure E17 Non-Capturing Pacemaker Competing with Supraventricular Rhythm in First Part of Strip,
Converting to Ventricular Tachycardia Competing with Pacemaker at End of Strip
Pacemakers 543
Figure E18 Ventricular Demand Pacemaker (with Underlying Atrial Fibrillation)
Figure E19 Ventricular Pacemaker Showing 100% Capture
544
ACLS: Advanced cardiac life support.
AF: Atrial Flutter.
Af: Atrial Fibrillation.
Atrial Kick: The final push during the last stage of atrial
contraction, during which blood flow increases as the
atrium empties. Loss of atrial kick can reduce cardiac
output by 20–30%.
AMI: Acute myocardial infarction.
ANS: Autonomic nervous system.
Anterior MI: Infarction of the anterior wall of the heart,
most often caused by occlusion of the Left Anterior
Descending coronary artery.
Anterior Surface: The plane of the heart that faces forward,
abutting the chest wall.
Anterobasal MI: Anterior wall infarction localized to the
base (top) of the heart.
Anterolateral MI: Anterior wall infarction localized to the
area of the lateral wall.
Anteroseptal MI: Anterior wall infarction localized to the
area of the septum.
Aorta: Artery that carries oxygenated blood from the left
ventricle to distal parts of the body.
Aortic Valve: The valve that controls passage of blood from
the left ventricle to the aorta.
Apex: The lower point of the heart.
Apical MI: Infarction localized to the apex (bottom) of the
heart.
Arrhythmia: The graphic representation of the heart’s
electrical activity; the term is loosely used to mean an
abnormality of the heart’s electrical function, but it is
also used to categorize patterns of electrical activity, not
all of which are necessarily abnormal or bad; also called
dysrhythmia; it can also be used to mean that a rhythm is not
regular.
Artifact: Electrical activity displayed on graph paper
that is superimposed on cardiac tracings, interfering with
interpretation of the rhythm; can be caused by outside
electrical sources, muscle tremors, patient movement; also
called interference.
Asystole: The absence of any cardiac electrical activity;
appears as a straight line on graph paper.
AT: Atrial Tachycardia.
Atria: The upper two chambers of the heart.
Atrial Arrhythmia: A cardiac arrhythmia originating from
the conduction system within the atria.
Atrial Fibrillation (Af): The cardiac arrhythmia in which
the atria are controlled by numerous irritable foci, thereby
causing ineffectual, chaotic atrial activity and irregular
ventricular response.
Atrial Flutter (AF): The cardiac arrhythmia in which an irritable focus in the atria produces a rapid, repetitive discharge,
resulting in rhythmic atrial depolarizations at a rate of
250–350 bpm, some of which are usually blocked by the AV
node to keep the ventricular rate in a more normal range.
Atrial Hypertrophy: Enlargement of myocardial wall in one
or more of the atria.
Atrial Kick: The increased pressure of atrial contraction
immediately before ventricular contraction; this “priming”
force increases ventricular efficiency, and can account for up
to 30% of cardiac output.
Atrial Pacemaker: An electrical stimulus originating in
the atria that paces the heart; an artificial pacemaker that
stimulates only the atria.
Atrial Tachycardia (AT): The cardiac arrhythmia in which
a single irritable focus in the atria takes over control of the
heart to produce a rate of 150–250 bpm; this arrhythmia
is often paroxysmal in nature—that is, it starts and stops
suddenly; in that instance it is called Paroxysmal Atrial
Tachycardia (PAT).
Atrioventricular Dissociation: A conduction defect that
causes the atria and ventricles to depolarize and function
independently; AV Dissociation.
Augmented Leads: Unipolar leads that measure electrical
flow on the frontal plane from the center of the heart to each
of three limb electrodes: Leads aVR, aVL, and aVF.
Automaticity: The unique ability of cardiac pacemaker cells
to initiate spontaneous excitation impulses.
Autonomic Nervous System: The system responsible for
control of involuntary bodily functions, including cardiac
and vascular activity; branches are sympathetic nervous
system and parasympathetic nervous system.
AV: Atrioventricular.
aVF: Augmented voltage, foot (left leg).
AV Heart Block: Arrhythmias caused by disturbances in
conduction through the AV node.
AV Junction: That part of the cardiac conduction system
that connects the atria and the ventricles; contains the AV
node and the non branching part of the Bundle of His.
aVL: Augmented voltage, left.
AV Node: A part of the cardiac conduction system located
within the AV junction; does not contain pacemaking cells;
its purpose is to slow conduction of impulses through the
AV junction.
aVR: Augmented voltage, right.
AV Sequential Pacemaker: A type of artificial pacemaker
that senses ventricular activity and, in its absence, paces
atria and ventricles in a normal sequence.
AV Synchronous Pacemaker: A type of artificial pacemaker
that senses both atria and ventricles and paces the ventricles
when a spontaneous P wave is not followed by a QRS.
Glossary
Glossary 545
The ventricles are depolarized in synchrony with P waves so
that atrial and ventricular contractions are coordinated.
Axis: Sum direction of electrical flow through the heart.
The axis of a given lead is the lead axis. The sum direction
of electrical flow through the heart as a whole is the mean
QRS axis.
Axis Deviation: Shift in mean QRS axis reflecting
myocardial damage, enlargement, or conduction defect.
Base: The upper end of the heart.
Baseline: The isoelectric line; the line on EKG graph paper
that indicates lack of electrical activity and from which all
other cardiac wave impulses deviate.
BBB: Bundle branch block.
Bigeminy: A pattern of cardiac electrical activity in which
every other beat is an ectopic, usually a PVC.
Biological Death: The second phase of death, following
clinical death; defined by brain death, usually following
4–6 minutes of cardiac arrest if no resuscitation is instituted.
Biphasic: A single EKG wave that has two deflections, one
upright and the other inverted.
Bipolar Lead: A lead composed of one positive and one
negative electrode.
Block: A defect in conduction within the heart’s electrical
system.
Bolus: A single loading dose of a drug; used to achieve a
rapid high therapeutic blood level prior to instituting IV drip
therapy.
bpm: Beats per minute.
Bradyarrhythmia: Any cardiac arrhythmia with a rate below
60 bpm.
Bradycardia: A heart rate less than 60 bpm.
Bundle Branches: The portion of the cardiac conduction
system within the ventricles that conducts impulses from the
Bundle of His to the Purkinje fibers; consists of right and left
bundle branches.
Bundle Branch Block (BBB): A conduction disturbance that
prevents or delays passage of impulses from the Bundle of
His through to the Purkinje network; can involve the right or
left bundle branch or, less frequently, both.
Bundle of His: That part of the cardiac conduction system
that conducts impulses from the AV Junction through to the
bundle branches.
Ca: Calcium.
CAD: Coronary artery disease.
Calibration: The act of standardizing the graphic display of
electrical activity; the calibration mark should measure
1 millivolt on the graph paper.
Capture: The heart responding to an electrical stimulus with
depolarization; generally refers to an arrhythmia’s response
to an artificial pacemaker.
Cardiac Arrest: The cessation of cardiac function, resulting
in sudden drop in perfusion and resultant clinical death.
Cardiac Cycle: The interval from the beginning of one
heartbeat to the beginning of the next; on the EKG it
encompasses the PQRST complex.
Cardiac Output (CO): The amount of blood pumped by the
left ventricle in one minute; it is calculated by multiplying
the stroke volume by the heart rate and is measured in liters
per minute.
Cardioversion: A maneuver used to convert various
tachyarrhythmias to more viable rhythms; consists of
application of electrical countershock (DC current) to the
chest wall; the electrical discharge is usually synchronized
to fall on the R wave, thus avoiding the relative refractory
period.
Carotid Sinus Massage: A maneuver used to convert
various supraventricular tachycardias to a more viable
rhythm; consists of gentle massage with fingertips over the
carotid sinus in the neck.
Central Terminal: An electrically neutral reference point
created by combining two or more electrodes. It is used to
oppose the positive electrode in unipolar leads.
CHB: Complete Heart Block.
CHF: Congestive Heart Failure.
Chordae Tendineae: Specialized fibers that connect heart
valve leaflets to papillary muscles to prevent backflow
during contractions.
Classical Second-Degree Heart Block: The term formerly
used to describe the type of AV heart block that is now called
Type II Second-Degree Heart Block, in which the AV node
intermittently blocks sinus impulses, preventing them from
being conducted through to the ventricles.
Clinical Death: The absence of pulse and blood pressure;
occurs immediately following cardiac arrest.
CO: Cardiac output.
Compensatory Pause: The time lag following an ectopic beat
before the next normal beat occurs; is identified by measuring
the interval from the R wave immediately preceding the
ectopic to the R wave immediately following it; a fully
compensatory pause (such as occurs following most PVCs)
will be exactly two times the normal R–R interval.
Competition: Condition in which an artificial pacemaker
competes with the patient’s intrinsic pacemaker for control
of the heart.
Complete Heart Block (CHB): Third-Degree AV Block; a
form of AV dissociation.
Conduction Ratio: The number of P waves to QRS
complexes. One P wave for every QRS complex is a 1:1
conduction ratio. Three P waves for every QRS complex
would be a 3:1 conduction ratio.
Conduction System: The pathways of conductive tissues
within the heart that facilitate passage of electrical impulses
throughout the myocardium.
Conductivity: The property of some cardiac cells that
enables them to transmit electrical impulses.
Contractility: The ability of heart muscle to contract in
response to electrical stimulation.
Conventional 12-Lead EKG: Electrocardiogram that
provides images of cardiac electrical activity from 12
perspectives simultaneously.
Coronary Arteries: Those blood vessels that supply the
heart muscle itself. The major branches are the Left Coronary
546 Glossary
Artery (with the Left Anterior Descending branches and the
Circumflex branch) and the Right Coronary Artery.
Coronary Sinus: The reentry point within the right atrium
where deoxygenated blood returns after having supplied the
heart muscle itself.
CPR: Cardiopulmonary resuscitation.
CT: Central terminal.
DDD: Double paced, double sensed, double response
(optimal pacemaker).
Defibrillation: Application of electrical countershock
(DC current) to the chest wall to terminate ventricular
tachyarrhythmias.
Demand Pacemaker: Pacemaker that senses patient’s
intrinsic complexes and fires only when needed.
Depolarization: The electrical process of discharging
polarized cells, usually resulting in muscle contraction.
Diaphragmatic Surface: The plane of the heart that faces
downward, resting against the diaphragm.
Diastole: The phase of the cardiac cycle in which chambers
are relaxing.
Digitalis Toxicity: Excessive blood levels of the heart drug
digitalis; also called dig-toxicity.
Dissociation: Independent function of two parts, generally
the atria and ventricles.
Dual-Chamber Pacemaker: Paces both atria and ventricles.
DVI: Double paced, ventricle sensed, inhibited response (AV
sequential pacemaker).
Dysrhythmia: Arrhythmia.
ECG: Electrocardiogram; see EKG.
Ectopic: Originating from a focus other than the primary
pacemaker.
Einthoven’s Triangle: The triangle created by an imaginary
line connecting the three chest electrodes used to create the
standard limb leads.
EKG: Electrocardiogram; electrocardiograph.
Electrocardiogram (EKG; ECG): Graphic representation of
the electrical activity in the heart.
Electrode: Metal wire attached to the patient’s body for the
purpose of conveying electrical impulses to a machine for
recording or displaying.
EMD: Electromechanical dissociation.
Endocardium: The inner layer of the heart wall, which
contains the branches of the heart’s electrical system.
Epicardium: The outside layer of the heart wall, which
contains the coronary blood vessels and nerves.
Equiphasic: A single EKG wave that has two deflections of
equal magnitude, one upright and the other inverted.
ERAD: Extreme right axis deviation.
Escape: The mechanism that allows a lower pacemaker
site to assume pacemaking responsibilities when a higher
site fails.
Evolving Infarction: The changing EKG picture associated
with the passage of time following myocardial infarction.
Failure to Capture: Situation in which the pacemaker fires
but the patient’s heart does not respond with depolarization.
Fibrillation: Chaotic, ineffective movement of the heart muscle.
First-Degree Heart Block: A type of AV heart block
characterized by prolonged but consistent conduction of
atrial impulses through to the ventricles.
Fixed-Rate Pacemaker: Pacemaker that is set at a
predetermined rate and fires regardless of the patient’s
underlying rhythm.
Flutter: Rhythmic, rapid beating of the heart muscle.
F waves: Flutter waves.
f waves: Fibrillatory waves.
Gallop Rhythms: Heart sounds that are grouped together so
they sound like galloping horses.
Heart Block: AV heart block.
Heart Rate (HR): The number of heart beats per minute.
Heart Sounds: Sounds associated with flow of blood
through heart chambers and closing of heart valves. The four
components are S1: closure of mitral and tricuspid valves;
S2: closure of aortic and pulmonic valves; S3: abnormally
rapid ventricular filling; S4: abnormally forceful atrial
contraction.
His–Purkinje System: The lower part of the cardiac
conduction system that transmits impulses throughout
the ventricles; located in the interventricular septum and
ventricular walls.
HR: Heart rate.
Hyperkalemia: High blood potassium level.
Hypertrophy: Enlargement of myocardial wall in one or
more of the chambers of the heart.
Hypokalemia: Low blood potassium level.
IAS: Interatrial septum.
Idioventricular Rhythm: A ventricular escape rhythm;
characterized by a rate less than 40 bpm.
Impulse Formation: The process by which cardiac electrical
cells create an electrical impulse without stimulation from
another source. See Automaticity.
Inferior MI: Infarction of the inferior wall of the heart, most
often caused by occlusion of the right coronary artery.
Inferior Vena Cava (IVC): Vein that carries deoxygenated
blood from the lower body back to the right atrium.
Interference: See Artifact.
Interpolation: The placement of an ectopic (especially PVCs)
between two normal beats without disturbing the regularity
of the underlying rhythm.
Interval: Distance between two points on an EKG tracing.
Interventricular Septum (IVS): The muscular wall dividing
the right and left ventricles.
Intraatrial Pathways: Branches of the cardiac conduction
system that service the atria.
Intraventricular Conduction Defect (IVCD): Disturbance
in conduction involving one or more of the bundle branches.
Also called Bundle Branch Block.
Glossary 547
Intrinsicoid Deflection: Reflection of the time it takes for peak
voltage to develop within the ventricles; measured from the onset
of the QRS to the peak of the R wave; a prolonged intrinsicoid
deflection (> 0.06 sec) is considered a “late R wave.”
Ischemic Changes: Changes on the EKG that reflect
myocardial ischemia: ST elevation, ST depression, Q wave
deepening, and T wave inversion.
Isoelectric Line: The line created on EKG graph paper when
no electrical current is flowing; see also Baseline.
IV: Intravenous.
IVC: Inferior vena cava.
IVCD: Intraventricular conduction defect.
IVS: Interventricular septum.
J Point: The demarcation between the end of the QRS
complex and the beginning of the ST segment.
JT: Junctional Tachycardia.
Junction: See AV Junction.
Junctional Escape Rhythm: An arrhythmia resulting from
failure of a higher pacemaker site, allowing the AV junction
to pace the heart at a bradycardia rate.
Junctional Tachycardia (JT): A rapid arrhythmia originating
in the AV junction.
K: Potassium.
LA: Left atrium; left arm.
LAD: Left axis deviation; left anterior descending branch of
the left coronary artery.
LAE: Left atrial enlargement.
Lateral MI: Infarction of the lateral wall of the heart, most
often caused by occlusion of the Left Anterior Descending
coronary artery.
Lateral Surface: The plane of the heart that faces the side,
just above the diaphragmatic surface.
LBB: Left bundle branch.
LBBB: Left bundle branch block.
LCA: Left coronary artery.
Lead: An electrocardiographic view of the heart, gained
by recording the electrical activity between two or more
electrodes.
LV: Left ventricle.
LVE: Left ventricular enlargement.
MCL1: Modified Chest Left; a monitoring lead that mimics
V1 and is useful in differentiating tachycardias.
MI: Myocardial Infarction.
Millivolts (mV): A measure of electricity; 1 volt equals
1,000 millivolts.
Mitral Valve: The valve that controls passage of blood from
the left atrium to the left ventricle.
mm: Millimeter.
Monitor: The machine on which electrocardiographic
impulses are displayed; oscilloscope.
Monitoring Lead: A lead that clearly shows individual wave
forms and is useful for monitoring cardiac rhythm, most
often Lead II or MCL1.
Multifocal: Term used to describe ectopic beats that
originate from more than one irritable focus. Also called
Multiformed.
Murmurs: Heart sounds caused by abnormal turbulence
associated with high flow rates, damaged valves, dilated
chambers, or backward flow.
mV: Millivolt.
Myocardial Infarction (MI): Tissue death caused by lack of
oxygen to the myocardium.
Myocardial Injury: Heart tissue damage caused by
sustained lack of oxygen.
Myocardial Ischemia: Initial heart tissue response to lack of
oxygen.
Myocardium: The center layer of the heart wall, consisting
of cardiac muscle fibers.
Na: Sodium.
Noise: Electrical interference displayed on graph paper that
interferes with interpretation of the underlying arrhythmia;
see Artifact.
Non-Q Infarction: Myocardial infarction that fails to
produce classic Q wave changes, most often because
the infarcted area is limited to partial thickness of the
myocardium; subendocardial infarction.
Normal Sinus Rhythm (NSR): The usual cardiac electrical
pattern of healthy people.
NSR: Normal Sinus Rhythm.
Oscilloscope: Display device with a screen for viewing EKG
and other physiological information; monitor.
PAC: Premature Atrial Complex.
Pacemaker: The source of electrical stimulation for cardiac
rhythm.
Pacemaker, Artificial: A device used to provided artificial
electrical stimuli to myocardial tissue to cause myocardial
depolarization.
Pacemaker Electrode: The conducting wire that connects to
the myocardium to deliver the pacemaker stimulus.
Pacemaker Site: The site of origin of the electrical
stimulation that is causing the cardiac rhythm.
Palpitations: The feeling the patient senses when the heart is
beating abnormally.
Papillary Muscles: Specialized muscles in the ventricles that
attach to heart valves by way of chordae tendineae, enabling
the valves to open and close.
Parasympathetic Nervous System: A branch of the
autonomic nervous system involved in control of
involuntary bodily functions; depresses cardiac activity
in opposition to the sympathetic branch of the ANS;
effects include slowing of heart rate and conduction and
diminished myocardial irritability.
Paroxysmal: Sudden onset and cessation; often used to
describe Atrial Tachycardia if it is characterized by abrupt
onset and termination.
Paroxysmal Atrial Tachycardia (PAT): The term used to
describe an Atrial Tachycardia characterized by abrupt onset
and cessation.
548 Glossary
PAT: Paroxysmal Atrial Tachycardia.
PEA: Pulseless Electrical Activity. A condition in which the
EKG shows a viable rhythm, but no pulse is detectible in the
patient. The electrical activity in the heart is not producing a
mechanical response (pulse).
Pericarditis: Inflammation of the pericardial sac
surrounding the heart. Causes ischemic changes on EKG and
can thus be misinterpreted to be MI.
Pericardium: A thin layer of tissue that forms the pericardial
sac to encase the heart in lubricating fluid.
Permanent Pacemaker: Pacemaker that is surgically
implanted within the patient’s body for an extended time.
PJC: Premature Junctional Complex.
Posterior Surface: The plane of the heart that faces
backward, abutting the spine.
PQRST: A single cardiac cycle on the EKG graph paper;
includes the P wave, QRS complex, and T wave and any
segments and intervals between.
Precordial Leads: Leads that measure electrical flow on the
horizontal plane, from the center of the heart to locations
around the anterior and lateral chest walls. The V leads: V1,
V2, V3, V4, V5, and V6.
Premature Atrial Complex (PAC): An ectopic beat
originating from an irritable focus in the atria.
Premature Junctional Complex (PJC): An ectopic beat
originating from an irritable focus in the AV Junction.
Premature Ventricular Complex (PVC): An ectopic beat
originating from an irritable focus in the ventricles.
PR Interval (PRI): The time interval on EKG graph paper
measured from the beginning of the P wave to the beginning
of the R wave; includes both the P wave and the PR segment;
indicates time of atrial depolarization.
PRI: P–R interval.
PR Segment: The time interval on EKG graph paper
measured from the end of the P wave to the beginning of the
R wave; indicates delay in the AV node.
Pulmonary Artery: Artery that carries deoxygenated blood
from the right ventricle to the lungs.
Pulmonary Vein: Vein that carries oxygenated blood from
the lungs back to the left atrium.
Pulmonic Valve: The valve that controls passage of blood
from the right ventricle to the pulmonary artery.
Pulse Deficit: The situation in which the heart is contracting,
but not all of the pulsations are reaching the periphery; the
difference between heart beats heard with a stethoscope and
the pulse rate measured at the periphery.
Pulseless Electrical Activity: A condition in which the EKG
shows a viable rhythm, but no pulse is detectible in the
patient. The electrical activity in the heart is not producing a
mechanical response (pulse).
Pulse Generator: The power source (battery unit) that drives
an artificial pacemaker.
Pump (Sodium): See Sodium Pump.
Purkinje System: The part of the cardiac conduction system
that transmits impulses from the bundle branches to the
myocardial cells in the ventricles; consists of Purkinje fibers
and terminal branches.
PVC: Premature Ventricular Complex.
P Wave: The first wave form in the normal cardiac cycle;
indicates atrial depolarization.
QRS Complex: The wave form on an EKG that represents
ventricular depolarization; includes the Q, R, and
S waves.
QT Interval: The time interval from the beginning of the
QRS complex to the end of the T wave; varies with
heart rate.
Quadrigeminy: A cardiac rhythm in which ectopics replace
every fourth normal beat, resulting in a cycle of three normal
beats and one ectopic, repeated continuously.
Q Wave: The first negative deflection following the P wave,
but before the R wave.
RA: Right atrium; right arm.
RAD: Right axis deviation.
RAE: Right atrial enlargement.
RBB: Right bundle branch.
RBBB: Right bundle branch block.
RCA: Right coronary artery.
Reciprocal Changes: EKG deflections seen in leads that
are opposite of each other and thus are mirror images.
For example, ST elevation in a facing lead would be ST
depression in an opposite lead. Reciprocal changes in
anterior leads are used to locate posterior infarctions.
Refractory: The state wherein the electrical cells are unable
to respond to electrical stimulation because they have not yet
recovered from the previous discharge.
Refractory Period: That portion of the cardiac cycle in
which the heart is unable to respond to electrical stimulation
because it has not yet recovered from the preceding
depolarization; consists of the Absolute Refractory Period
(QRS complex and upslope of the T wave) and the Relative
Refractory Period (downslope of the T wave).
Relative Refractory Period: The terminal portion of the
cardiac refractory period, during which a strong enough
electrical stimulus could discharge the heart, resulting in
inefficient and potentially dangerous arrhythmias; located
on the downslope of the T wave.
Repolarization: The process of recharging depolarized cells
back to their “ready” (polarized) state.
Retrograde Conduction: Electrical current that arises from
the area of the AV junction and travels backward up toward
the SA node to depolarize the atria in the opposite direction
of normal.
Rhythm: The regularity of a cardiac pattern; generally used
to refer to the arrhythmia itself, rather than its rhythmicity
(i.e., “The patient’s rhythm is Atrial Fibrillation”), even
though Atrial Fibrillation is an irregular arrhythmia;
synonymous with arrhythmia.
“R on T” Phenomenon: The situation in which the R wave of
a PVC occurs on or near the downslope of the preceding
T wave, thereby falling in the vulnerable phase of the cardiac
cycle, the Relative Refractory Period, and threatening to cause
Glossary 549
premature discharge and result in an ineffective pattern, such
as Ventricular Tachycardia or Ventricular Fibrillation.
Runaway Pacemaker: A malfunction of an artificial
pacemaker that causes it to fire at an excessive rate.
RV: Right ventricle.
RVE: Right ventricular enlargement.
R Wave: The first upright deflection following the P wave, or
the first positive wave of the QRS complex.
S/S: Signs/symptoms.
SA: Sino-atrial.
SA: Sinus Arrhythmia.
SB: Sinus Bradycardia.
sec: Second.
Second-Degree Heart Block, Type I: A type of SecondDegree Heart Block in which sinus impulses are delayed at
the AV node for increasingly long periods, until conduction
is blocked completely, then the cycle repeats itself;
Wenckebach. (Formerly called Mobitz Type I.)
Second-Degree Heart Block, Type II: A type of SecondDegree Heart Block in which the AV node selectively
blocks some beats while allowing others to pass through
to the ventricles. The EKG shows more P waves than
QRS complexes; some P waves are not followed by QRS
complexes. (Formerly called Classical Second-Degree Heart
Block, or Mobitz Type II.)
Septum: The wall that divides the heart into right and left
sides. The thin wall between the atria is called the interatrial
septum. The thicker wall between the ventricles is the
interventricular septum.
Single-Chamber Pacemaker: Paces only the atria or the
ventricles, but not both.
Sinus Arrhythmia (SA): The arrhythmia in which the
pacemaker is located in the SA node but discharges
irregularly; usually correlated with respirations; rate
increases on inspiration, and decreases on expiration.
Sinus Bradycardia (SB): The arrhythmia in which the
pacemaker is located in the SA node but discharges at a rate
less than 60 bpm.
Sinus Node: The normal pacemaker of the heart; located at
the junction of the superior vena cava and the right atrium;
SA node; sinoatrial node.
Sinus Rhythm: Any rhythm that originates in the sinus (SA)
node; used loosely to refer to Normal Sinus Rhythm.
Sinus Tachycardia (ST): The arrhythmia in which the
pacemaker is located in the SA node but discharges at a rate
greater than 100 bpm.
Sodium Pump: The chemical phenomenon that takes place
at a cellular level within the cardiac electrical conduction
system, in which sodium and potassium trade places across
the cell wall, thereby initiating the flow of electrical current
within the heart.
Standardization: The act of calibrating the EKG machine or
oscilloscope to a standard (1 millivolt).
Standard Limb Leads: The bipolar leads showing the frontal
plane: Leads I, II, and III.
Stroke Volume: The amount of blood ejected with each
contraction of the left ventricle.
ST: Sinus Tachycardia.
STEMI: ST Elevation Myocardial Infarction.
ST Segment: The portion of the cardiac cycle between the S
wave and the T wave.
Subendocardial Infarction: Infarcted area too small to
extend all the way through the ventricular wall; also called
a less-than-transmural infarction, or non-Q infarction, since
the damage is often insufficient to create the classic Q wave
changes seen with larger infarctions.
Superior Vena Cava (SVC): Vein that carries deoxygenated
blood from the upper body back to the right atrium.
Supraventricular (SV): Originating above the ventricles.
Supraventricular Tachycardia (SVT): Term used to describe
a rapid arrhythmia that is regular, has no visible P waves,
and has a rate range common to other arrhythmias, thereby
making more accurate identification impossible; commonly
applied to Atrial Tachycardia, Junctional Tachycardia, Sinus
Tachycardia, and Atrial Flutter with 1:1 response; loosely
used to refer to any tachycardia that originated above the
ventricles.
SV: Supraventricular.
SVC: Superior vena cava.
SVT: Supraventricular tachycardia.
S Wave: The second negative deflection following the P
wave, or the first negative deflection following the R wave.
Sympathetic Nervous System: One of the two main
branches of the autonomic nervous system, which controls
involuntary bodily functions; stimulates cardiac activity
in opposition to the parasympathetic branch; effects
include increased heart rate and conduction and increased
myocardial irritability.
Systole: The phase of the cardiac cycle in which the
chambers are contracting.
Tachyarrhythmia: Any cardiac arrhythmia with a
ventricular rate greater than 100 bpm.
Tachycardia: Heart rate greater than 100 bpm.
Temporary Pacemaker: An artificial pacemaker used in
acute setting to stabilize and maintain patient for short
periods.
Third-Degree Heart Block: The arrhythmia in which all
atrial impulses are prevented from reaching the ventricles
because of a complete block at the AV node; constitutes a
form of AV dissociation because atria and ventricles function
totally independent of each other; Complete Heart Block
(CHB).
Transcutaneous Pacemaker: Pacemaker stimulus delivered
across the skin via external pads.
Transmural Infarction: Infarcted area large enough to
extend completely through the wall of the ventricle.
Transvenous Pacemaker: Pacemaker wire inserted through
a vein.
Tricuspid Valve: The valve that controls passage of blood
from the right atrium to the right ventricle.
550 Glossary
Trigeminy: A pattern in which ectopics occur every third
beat, producing a repetitive cycle of two normal beats and
one ectopic.
Unifocal: Refers to ectopic beats that originate from a single
irritable focus. Also called Uniformed.
Uniphasic: A single EKG wave that has only one phase,
either upright or inverted.
Unipolar Lead: A lead that has only one charged electrode
(positive electrode). The opposing pole is created by
combining other electrodes into an electrically neutral
reference point (the central terminal).
U Wave: Low-voltage wave following the T wave, having
the same polarity as the T wave. Usually not apparent but
becomes more pronounced in hypokalemia.
Variable Conduction: A changing conduction ratio within
a given strip. For example, rather than having a consistent
ratio of 2 Ps for every QRS complex (a 2:1 ratio) across an
entire strip, a variable conduction would show 2:1, then 3:2,
then back to 2:1, or maybe 4:3, all within the same strip.
VDD: Ventricle paced, double sensed, double response (AV
synchronous pacemaker).
Vector: Direction of flow of cardiac electrical activity;
represented by an arrow with the point indicating the positive
pole and the size of arrow indicating magnitude of current.
Ventricles: The lower two chambers of the heart.
Ventricular Depolarization: Discharge of electrical
activity throughout the ventricles to stimulate ventricular
contraction; produces the QRS complex on an EKG.
Ventricular Fibrillation (VF): The arrhythmia in which
the ventricles are controlled by numerous irritable foci,
producing chaotic, ineffective muscle activity rather than the
normal contraction.
Ventricular Flutter (VF): The arrhythmia in which a single
irritable focus in the ventricles depolarizes the heart at a rate
greater than 250 bpm; usually considered to be a rapid form
of Ventricular Tachycardia.
Ventricular Hypertrophy: Enlargement of myocardial wall
in one or more of the ventricles.
Ventricular Pacemaker: An electrical stimulus from the
ventricles that controls heart rhythm; an artificial pacemaker
that stimulates only the ventricles.
Ventricular Standstill: The arrhythmia in which the
ventricles are not depolarized by any electrical stimulation
and therefore do not contract; if atrial activity is present, it
is not conducted through the AV node; if no atrial activity is
present, it is called Asystole.
Ventricular Tachycardia (VT): The arrhythmia in which a
single irritable focus in the ventricles depolarizes the heart at
a rate of 150–250 bpm.
VF: Ventricular Fibrillation, Ventricular Flutter.
VT: Ventricular Tachycardia.
Vulnerable Period: The period in the cardiac cycle when
the heart is most susceptible to premature discharge,
with a resultant ineffective pattern if it receives a strong
enough electrical stimulus; the Relative Refractory Period;
corresponds with the downslope of the T wave.
VVI: Ventricle paced, ventricle sensed, inhibited response
(ventricular demand pacemaker).
Wandering Pacemaker (WP): The arrhythmia in which the
pacemaker site shifts from the SA node to the atrium and
back again, sometimes dropping as low as the AV junction.
Waves: Deflections on the electrocardiograph caused by
changes in electrical activity in the heart.
Wenckebach: The form of Second-Degree AV Block in which
the node progressively holds each impulse longer until one
is eventually not conducted, then the cycle starts over; Type I
Second-Degree Heart Block.
WP: Wandering Pacemaker.
551
Chapter 2,
Practice Strips Answers
PART I: LABELING WAVES
Answers Key
2.1
P
S
T
R
2.2
P T
S
R
2.3
R
T
S
Q
P
2.4
R
P
Q
T
2.5
P T
QS
2.6
P
R
T
S
552 Answers Key
2.7
P R T
Q S
2.8
R
T P
2.9
R
T P
Q
2.10
R
T
P
S Q
2.11
T R P
Q S
2.12
R
T
P
Q S
Answers Key 553
Chapter 3
Regularity: regular
Rate: 79 bpm
P Waves: regular
P–P interval; uniform
waves
PRI: 0.16 second and
constant
QRS: 0.08 second
Regularity: regular (very
slightly irregular)
Rate: approximately 90 bpm
P Waves: uniform; regular
P–P interval
PRI: 0.12 second and
constant
QRS: 0.08 second
Regularity: regular
Rate: 63 bpm
P Waves: uniform; regular
P–P interval
PRI: 0.16 second and
constant
QRS: 0.10 second
3.1
3.2
3.3
Regularity: regular
Rate: 125 bpm
P Waves: uniform, regular
P–P interval
PRI: 0.16 second and
constant
QRS: 0.08 second
3.4
Regularity: regular
Rate: 83 bpm
P Waves: uniform; regular
P–P interval
PRI: 0.16 second and
constant
QRS: 0.12 second
3.5
Regularity: regular
Rate: 107 bpm
P Waves: uniform; regular
P–P interval
PRI: 0.20 second and
constant
QRS: 0.10 second
3.6
Regularity: regular
Rate: 79 bpm
P Waves: uniform; regular
P–P intervals
PRI: 0.10 second and
constant
QRS: 0.06 second
3.7
Regularity: regular
Rate: 79 bpm
P Waves: uniform; regular
P–P interval
PRI: 0.16 second and
constant
QRS: 0.10 second
3.8
Regularity: regular
Rate: 83 bpm
P Waves: uniform; regular
P–P interval
PRI: 0.20 second and
constant
QRS: 0.08 second
3.9
Regularity: irregular
Rate: approximately
80 bpm
P Waves: uniform; irregular
P–P interval
PRI: 0.16 second and
constant
QRS: 0.10 second
3.10
Regularity: regular (very
slightly irregular)
Rate: 83 bpm
P Waves: upright and
uniform
PRI: 0.14 second
QRS: 0.10 second
3.11
Regularity: regular
Rate: 50 bpm
P Waves: upright and
uniform
PRI: 0.12 second
QRS: 0.08 second
3.12
Regularity: regular
Rate: 79 bpm
P Waves: upright and
uniform
PRI: 0.12 second
QRS: 0.08 second
3.13
Regularity: irregular
Rate: 70 bpm
P Waves: upright and
uniform
PRI: 0.16 second
QRS: 0.08 second
3.14
Regularity: regular (very
slightly irregular)
Rate: 79 bpm
P Waves: upright and
uniform
PRI: 0.16 second
QRS: 0.08 second
3.15
Chapter 4
Regularity: regular (slightly
irregular)
Rate: 48 bpm
P Waves: uniform and upright; regular P–P interval
PRI: 0.18 second and
constant
QRS: 0.12 second
Interp: Sinus Bradycardia
(with wide QRS)
4.1
Regularity: irregular
Rate: approximately 80 bpm
P Waves: uniform and upright; regular P–P interval
PRI: 0.14 second and
constant
QRS: 0.06 second
Interp: Sinus Arrhythmia
4.2
Regularity: regular
Rate: 75 bpm
P Waves: uniform and
upright; regular
P–P interval
PRI: 0.16 second and
constant
QRS: 0.08 second
Interp: Normal Sinus
Rhythm
4.3
Regularity: regular
Rate: 136 bpm
P Waves: uniform and upright; regular P–P interval
PRI: 0.16 second and
constant
QRS: 0.06 second
Interp: Sinus Tachycardia
4.4
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