compensatory
PVC
Figure 61 Mechanism of Premature Ventricular Complex
Rate: depends on
underlying rhythm
Pacemaker: an irritable
focus within the ventricles
Regularity: the ectopic
interrupts the regularity of
the underlying rhythm
Conduction: conduction
through the ventricles is
prolonged
A single irritable focus within the ventricles that fires prematurely to initiate an ectopic complex.
This is a single beat, not an entire rhythm; the underlying rhythm also must be identified.
Figure 62 T Wave Configuration in PVCs
(a)
T
QRS
(b)
T
QRS
Ventricular Rhythms 217
Figure 63 Examples of Typical PVC Configurations
(b)
X
X
(a)
(d)
X
(c)
X
(e)
X
(g) (h)
X
(f)
X
X
218 Chapter 8
2 times R-R interval
PVC with compensatory pause
Figure 64 Compensatory Pause
10. Although it is most common for PVCs to be followed by compensatory pauses, this
is not a rigid requirement. An alternative configuration possibility is that the PVC be
followed by no pause whatsoever. This occurs when the PVC squeezes itself in between
two regular complexes and does not disturb the regularity of the sinus node. This is
called an interpolated PVC, because the PVC inserts itself between two regular beats
(Figure 65). With an interpolated PVC, the R–R interval remains ,
since the PVC does not interrupt the sinus rhythm.
11. If a PVC is followed by a pause before the next sinus beat, leaving a distance
between the sinus beat before the PVC and the sinus beat following the PVC,
which measures exactly twice the normal R–R interval, you would call this a
pause. However, if the PVC falls between two sinus
beats, without interrupting the regularity of the underlying rhythm, you would call
this an PVC.
12. As with other types of ectopics, a PVC will interrupt the underlying rhythm.
In interpreting the rhythm strip, it is important to identify both the ectopic and the
underlying arrhythmia. For example, the arrhythmia might be Sinus Tachycardia with
a PVC. When you are reporting a PVC, you should convey as much information about
the disorder as possible, including identifying the rhythm.
13. Several other items should be noted about a PVCs. Since PVCs are an indication
of myocardial irritability, it is important to note how frequently they are occurring.
If the patient is having only an occasional PVC, this may be a normal rhythm
regular
compensatory
interpolated
underlying
Figure 65 Interpolated PVC
1 times R-R interval
Interpolated PVC
Ventricular Rhythms 219
for that person. But if the frequency picks up so that you are seeing 5–10 per minute,
you would suspect that the ectopics were an indication of increasing myocardial
. If the patient is experiencing chest pain of any sort, even a
single PVC could be ominous. It is important that you note not only the presence of
PVCs, but also the of occurrence.
Unifocal vs. Multifocal
14. If a single focus within the ventricles is the source of the PVCs, all of these ectopics
will have an identical appearance. That is, if one PVC first has a positive deflection,
and then a wide negative deflection, all other PVCs from that single focus will have
that same configuration. These would be considered unifocal PVCs (Figure 66),
because they all came from a focus and are all uniform in
configuration. If all the PVCs on a rhythm strip had similar appearances, you would
assume that they all originated from a ectopic focus and would
call them PVCs.
15. In cases of greater irritability, several ventricular foci might begin to initiate
ectopics. In that case, the PVCs would have a variety of configurations (Figure 67).
If two foci were initiating PVCs, all of the PVCs would have one of two configurations.
irritability
frequency
single
single
unifocal
Figure 66 Unifocal PVCs
Figure 67 Multifocal PVCs
220 Chapter 8
If more sites were irritable, there would be a greater variety of .
When PVCs have multiple configurations, they are called multifocal because the heart
is so irritable that multiple foci are initiating the ectopics. Since they originate from
multiple foci, PVCs are more serious than unifocal PVCs
because they are associated with a more irritable myocardium.
16. If the PVCs on a rhythm strip all had the same basic configuration, they would
be considered . If the PVCs had differing configurations, they
would be considered .
The terms unifocal and multifocal are used to describe whether ectopics have a consistent
configuration or varying morphologies. These terms are based on the presumption that
uniform shapes result from a single irritable focus, whereas varying configurations are
caused by multiple irritable foci. You may hear other terms used for the same purpose:
uniformed for ectopics with one shape and multiformed for ectopics with varying shapes.
For our purposes, you may consider unifocal and uniformed to be synonymous. Consider
multifocal and multiformed synonymous as well.
R on T Phenomenon
17. PVCs represent a major electrical force, since they reflect a premature depolarization
of the ventricles. Because they come prematurely, they often fall near the end of the
preceding QRS complex. If you recall, in Chapter 2, we learned that a portion of
the T wave is considered a vulnerable area, because an electrical impulse falling
during that vulnerable phase could cause an aberrant depolarization of the heart. If a
PVC occurs during the phase, it could throw the heart into an
uncontrolled repetitive pattern. For this reason, it is important to note any PVC that falls
on or near the wave of the preceding beat. This phenomenon
is called R on T because the R wave of the PVC is hitting on the T wave of the preceding
complex (Figure 68). If you see PVCs creeping up on the preceding T wave, you would
call this an phenomenon and know that it represents a very
serious situation.
18. The R on T phenomenon exists when the R wave of a falls
on or near the vulnerable phase of the cardiac cycle. The vulnerable phase, or relative
refractory period, is located on the downslope of the wave.
(Note: If you have forgotten this material, turn back to Chapter 2, Frames 51–57, for a
quick review.)
configurations
multifocal
unifocal
multifocal
vulnerable
T
R on T
PVC
T
Figure 68 R on T Phenomenon: Ectopic A exhibits R on T phenomenon; Ectopic B does not
A B
Ventricular Rhythms 221
Runs and Couplets
19. Another sign of increasing myocardial irritability is when PVCs occur in immediate
succession, without a normal beat intervening. If only two PVCs occur before the
normal pattern resumes, you would see two PVCs attached to each other. This is called
a couplet (Figure 69), but if you see three or more PVCs occurring in succession, this
would be called a run of PVCs (Figure 70). The important distinction here is that several PVCs have fired without allowing the normal pacemaker to resume pacemaking
responsibility. This is an indication of significant . Regardless of
whether you call this pattern a couplet or a run of PVCs, you should note that the PVCs
are occurring in immediate succession and indicate the number of PVCs observed.
Technically, two successive PVCs would be called a , but it is
sufficient to call any number of successive PVCs a of PVCs and
then indicate the number of PVCs involved.
20. A pair of PVCs in immediate succession could be called either a
or a of two PVCs. But if there were
three or more PVCs in a row, it should be called a of three
(or more) . The important thing here is to note that an
increasing number of PVCs indicates increasing myocardial .
irritability
couplet
run
couplet
run
run
PVCs
Figure 69 PVCs Occurring as a Couplet (Pair)
Figure 70 PVCs Occurring in a Run
222 Chapter 8
Grouped Beating
21. Sometimes, frequently occurring PVCs will fall into a pattern with the surrounding
normal beats. This is called grouped beating. For example, you may see a PVC, then a
normal beat, then a PVC, then a normal beat, and so on. When the PVCs are falling in a
pattern of “every other beat” (Figure 71) with the normal beats, this is called bigeminy
(pronounced bī-jem’-eny). Bigeminy refers to a repetitive pattern of grouped beating
(e.g., one normal and one PVC) across the entire strip. When you see a pattern of one
PVC, then one normal beat, then one PVC, then one normal beat, and this pattern
continues across the strip, you would call the rhythm of PVCs. bigeminy
Figure 71 Patterns of Grouped Beating
(a)
Bigeminy
(b)
Trigeminy
(c)
Quadrigeminy
Ventricular Rhythms 223
22. Don’t forget, though, to include the identification of the underlying rhythm.
For example, if you saw a sinus beat, then a PVC, then a sinus beat, then a
PVC, across the strip, you would call this Sinus Rhythm with bigeminy of
.
23. Several other patterns of grouped beating that are very similar to bigeminy
result from PVCs falling into a rhythm with normal beats. For example, if you
saw a PVC followed by two sinus beats, then another PVC followed by two
sinus beats, you would have a repetitive cycle of three beats: one PVC, and
two sinus beats. This pattern is called trigeminy, since the cycle contains three beats.
Such a rhythm would be called Sinus Rhythm with
of PVCs.
24. Another such pattern is quadrigeminy, where a pattern of four beats consists of
one PVC and three normal beats. If you had a long enough rhythm strip, you could
probably map out patterns of as many as eight, nine, or more beats to a cycle.
However, the most common are bigeminy, a cycle consisting of one PVC and
one normal beat; trigeminy, a cycle including one PVC and two normal beats; and
, where there are four complexes to the cycle: one PVC,
and three normal beats.
25. Patterns such as bigeminy, trigeminy, and quadrigeminy can be found with other
ectopics as well as PVCs. For example, you can have bigeminy of PACs or quadrigeminy of PJCs. But to qualify as a true patterned beat, the grouping should continue
across the entire strip. Just because you happen to have two PACs on the strip with a
single normal beat between them, you could not necessarily call this bigeminy. But if
the pattern continued regularly across the strip, you would call it Sinus Rhythm with
bigeminy of .
26. You now know quite a few things about PVCs. They are wide and bizarre, with
a QRS measurement of . Frequently, the T wave will be in
the opposite direction of the . PVCs are a sign of myocardial
, so you should note how frequently they are occurring. You
should also note if they are all coming from a single focus, in which case you would
call them . If they are coming from more than one focus, you
would call them .
27. You should be very cautious with a PVC that is falling near the downslope of
the wave, since this is the vulnerable phase of the cardiac
cycle. This is called phenomenon and is dangerous because it
could throw the heart into an ineffective repetitive pattern.
28. If the myocardium is irritable enough, you may notice PVCs falling in succession,
without an intervening normal beat. If there were several PVCs connected in this
manner, you would call it a of PVCs and would note how
many ectopics were involved. If there were only two PVCs paired, you might call this
a .
29. Finally, you know that PVCs can fall into patterns with the underlying normal
beats. If it is a pattern of twos (i.e., one normal, one PVC), you would call this
. If it is a pattern of threes, it is called ,
and it is called quadrigeminy if there are complexes in
the pattern.
PVCs
trigeminy
quadrigeminy
PACs
0.12 second or more
QRS complex
irritability
unifocal
multifocal
T
R on T
run
couplet
bigeminy; trigeminy
four
224 Chapter 8
30. Here are the rules for PVCs (Figure 72):
Regularity: ectopics will disrupt regularity of underlying rhythm
Rate: depends on underlying rhythm and number of ectopics
P Waves: will not be preceded by a P wave; dissociated P wave may be seen
near PVC
PRI: since the ectopic comes from a lower focus, there will be no PRI
QRS: wide and bizarre; 0.12 second or greater; T wave is usually in opposite
direction from R wave
Ventricular Tachycardia (VT)
31. If the myocardium is extremely irritable, the ventricular focus could speed up and
override higher pacemaker sites. This would create what is essentially a sustained run
of PVCs. This rhythm is called Ventricular Tachycardia (VT) (Figure 73). In fact, a
run of PVCs is often called a short burst of VT. They both result from myocardial
, and they both fit the same rules. However, PVCs are
ectopics, whereas VT is an actual arrhythmia. In VT you will
see a succession of PVCs across the strip at a rate of about 150–250 bpm. This arrhythmia
usually has a very uniform appearance, even though the R–R interval may be slightly
irregular. It is possible for VT to occur at slower rates, but when it does, it is qualified
by calling it a slow VT. A true VT has a ventricular rate of bpm.
irritability
single
150–250
Figure 72 Rules for Premature Ventricular Complex
Premature Ventricular Complex
Regularity: The underlying rhythm can be regular or irregular. The ectopic PVC will interrupt the regularity of the
underlying rhythm (unless the PVC is interpolated).
Rate: The rate will be determined by the underlying rhythm. PVCs are not usually included in the rate
determination because they frequently do not produce a pulse.
P Waves: The ectopic is not preceded by a P wave. You may see a coincidental P wave near the PVC, but it is
dissociated.
PRI: Since the ectopic comes from a lower focus, there will be no PRI.
QRS: The QRS complex will be wide and bizarre, measuring at least 0.12 second. The configuration will
differ from the configuration of the underlying QRS complexes. The T wave is frequently in the opposite
direction from the QRS complex.
Ventricular Rhythms 225
32. Each of the other rules for a PVC also applies to VT. The QRS complex will be
0.12 second or greater, and the complex will be and bizarre,
with the T wave usually in the opposite direction of the wave.
Since this rhythm originates from a focus, you will not see
a P wave in front of the QRS complex. However, this is another form of AV dissociation, so you may see an occasional P wave coincidentally occurring near the QRS
complex. As with PVCs, VT will have a QRS measurement of
second or greater, with a bizarre configuration of the
complex and T wave, and there will be no waves preceding
the QRS complexes.
33. Ventricular Tachycardia is caused by a focus in the
ventricles that fires at a tachycardia rate to override the higher pacemaker sites
and take over control of the heart. It is also possible for the ventricular focus to
change to a flutter mechanism, which would result in an arrhythmia very similar to
VT but with a ventricular rate of more than 300. When the ventricles depolarize at
such a rapid rate, the resultant EKG pattern becomes a very uniform, regular tracing and looks almost like a coiled spring. There is very little difference between
VT and Ventricular Flutter, except for the rate. Most clinicians choose to consider
Ventricular Flutter in with VT and eliminate this academic distinction. For our
purposes, we will consider Ventricular Flutter a rapid form of VT. We will not make
a distinction between the two, since the only real difference is in the ventricular
.
34. Here are the rules of Ventricular Tachycardia (Figure 74):
Regularity: usually regular; can be slightly irregular
Rate: 150–250 bpm; can exceed 250 bpm if the rhythm progresses to
Ventricular Flutter; may occasionally be slower than 150 bpm,
in which case it is called slow VT
P Waves: will not be preceded by P waves; dissociated P waves may be seen
PRI: since the focus is in the ventricles, there will be no PRI
QRS: wide and bizarre; 0.12 second or greater; T wave is usually in opposite
direction from R wave
wide
R
ventricular
0.12
QRS
P
single
rate
Figure 73 Mechanism of Ventricular Tachycardia
Pacemaker: an irritable
focus within the ventricles
Rate: 150–250 bpm
Conduction: conduction
through the ventricles is
prolonged
Regularity: regular, can be
slightly irregular
An irritable focus within the ventricles fires regularly at a rapid rate to override higher sites for control
of the heart.
226 Chapter 8
Ventricular Fibrillation
35. In extreme myocardial irritability, the electrical foci in the ventricles can begin firing
in a rapid, disorganized manner. This rhythm is called Ventricular Fibrillation
(Figure 75). The ventricles can’t respond to such chaotic signals, and thus can’t pump
blood effectively. Ventricular Fibrillation is a lethal arrhythmia, because the heart’s
pumping function is totally .
36. Ventricular Fibrillation (VF) is probably the easiest of all the arrhythmias to
recognize. This is because there are no discernible complexes or intervals and the entire
rhythm consists of chaotic, irregular activity. Since there are no identifiable complexes
or wave forms, the EKG pattern of VF is simply a grossly
fibrillatory pattern.
ineffective
chaotic
Figure 74 Rules for Ventricular Tachycardia
Ventricular Tachycardia
Regularity: This rhythm is usually regular, although it can be slightly irregular.
Rate: Atrial rate cannot be determined. The ventricular rate range is 150–250 bpm. If the rate is below 150 bpm,
it is considered a slow VT. If the rate exceeds 250 bpm, it is called Ventricular Flutter.
P Waves: None of the QRS complexes will be preceded by P waves. You may see dissociated P waves
intermittently across the strip.
PRI: Since the rhythm originates in the ventricles, there will be no PRI.
QRS: The QRS complexes will be wide and bizarre, measuring at least 0.12 second. It is often difficult to
differentiate between the QRS and the T wave.
Figure 75 Mechanism of Ventricular Fibrillation
Pacemaker: multiple
irritable foci within the
ventricles
Rate: unable to determine
Conduction: prolonged to
the point that actual QRS
complexes are not
Regularity: grossly distinguishable
chaotic; no pattern of
electrical activity
Multiple foci within the ventricles become irritable and generate uncoordinated, chaotic impulses
that cause the heart to fibrillate rather than contract.
Ventricular Rhythms 227
37. VT is distinguishable from VF because Ventricular Tachycardia has wide, bizarre
complexes, but they are uniform and measurable. VF has no measurable waves
or .
38. Here are the rules for Ventricular Fibrillation (Figure 76):
Regularity:
Rate:
P Wave:
PRI:
QRS:
totally chaotic with no discernible waves or complexes
Idioventricular Rhythm
39. So far, you have learned three ventricular arrhythmias, all of which are the result
of ventricular irritability. These are PVCs, Ventricular Tachycardia, and Ventricular
Fibrillation. It is also possible for a ventricular rhythm to be produced by an escape
mechanism. If a higher pacemaker site fails, a ventricular focus can step in to take
over pacemaking responsibility. There are two ways a ventricular focus can assume
control of the heart. One is irritability and the other is
mechanism.
40. A ventricular escape rhythm is one that takes over pacemaking in the absence
of a higher focus and depolarizes the heart at the inherent rate of the ventricles, which
is bpm. This rhythm is called Idioventricular Rhythm
(Figure 77) because the ventricles are initiating the rhythm on their own, without a
conducted stimulus from a higher focus. The rate for Idioventricular Rhythm would
be bpm.
complexes
escape
20–40
20–40
Figure 76 Rules for Ventricular Fibrillation
Ventricular Fibrillation
Regularity: There are no waves or complexes that can be analyzed to determine regularity. The baseline is totally
chaotic.
Rate: The rate cannot be determined since there are no discernible waves or complexes to measure.
P Waves: There are no discernible P waves.
PRI: There is no PRI.
QRS: There are no discernible QRS complexes.
228 Chapter 8
41. You can consider Idioventricular Rhythm to be a ventricular escape rhythm, since
it is a fail-safe rhythm that takes over when pacemaker sites
fail. A ventricular focus firing within the inherent rate range of the ventricles would
produce a rhythm called Rhythm.
42. You should not see P waves in an Idioventricular Rhythm, since the escape mechanism would take over only if the supraventricular pacemaker sites had failed. What you
will see is a rhythm of very slow ventricular complexes, usually in a regular rhythm,
although it is possible for such an unreliable pacemaker to discharge irregularly. An
Idioventricular Rhythm will not have waves. Instead, you will
see ventricular complexes measuring at least second, firing at
a rate of 20–40 bpm.
43. Idioventricular Rhythm is initiated by the very last possible fail-safe mechanism
within the heart. This means that it is frequently an unreliable focus. It may fire a little
irregularly, and the rate may be less than 20 bpm, even though the intrinsic ventricular
rate is supposed to be 20–40 bpm. When the rhythm is in its terminal stages—that is, as
the patient is dying—the complexes can lose some of their form and be quite irregular.
In this stage, the arrhythmia is said to be agonal, or a dying heart. The word agonal is
used to describe a terminal, lethal arrhythmia, especially when it has stopped beating
in a reliable pattern. Idioventricular Rhythm is an rhythm,
especially when the rate drops below 20 bpm and the pattern loses its uniformity.
44. Here are the rules for Idioventricular Rhythm (Figure 78):
Regularity: usually regular
Rate: 20–40 bpm; can drop below 20 bpm
P Waves: none
PRI: none
QRS: wide and bizarre; 0.12 second or more
Asystole
45. The last stage of a dying heart is when all electrical activity ceases. This results in
a straight line on the EKG, an arrhythmia called Asystole (pronounced ā · siś-toe-lee)
(Figure 79). Asystole is a period of absent electrical activity, seen on the EKG as a
line, possibly with some undulations in it. Asystole is a lethal
arrhythmia that is very resistant to resuscitation efforts.
higher
Idioventricular
P
0.12
agonal
straight
Figure 77 Mechanism of Idioventricular Rhythm
Conduction: prolonged
conduction through the
ventricles
Pacemaker: escape focus
within the ventricles
Rate: usually 20-40 bpm,
but often falls below 20
bpm
Regularity: usually regular,
but can be regular at
slower rates
In the absence of a higher pacemaker, the ventricles initiate a regular impulse at their inherent rate to
take control of the heart.
Ventricular Rhythms 229
46. The absence of cardiac electrical activity will cause a straight line on the EKG. This
rhythm is called . To be certain there is no electrical activity,
you should first make sure the machine is not malfunctioning, then view the rhythm
in more than one lead.
Here are the rules for Asystole (Figure 80):
Regularity:
Rate:
P Wave:
PRI:
QRS:
straight line indicates no electrical activity
47. A straight line on the EKG would suggest that there is no electrical activity left in
the heart. This rhythm would be called .
Asystole
Asystole
Figure 78 Rules for Idioventricular Rhythm
Idioventricular Rhythm
Regularity: This rhythm is usually regular, although it is less reliable as the heart dies.
Rate: The ventricular rate is usually 20–40 bpm, but it can drop below 20 bpm.
P Waves: There are no P waves in this arrhythmia.
PRI: There is no PRI.
QRS: The QRS complex is wide and bizarre, measuring at least 0.12 second.
Figure 79 Mechanism of Asystole
Pacemaker: no pacemaker
is firing
Rate: no rate because no
pacemaker is firing
Conduction: no electrical
activity
Regularity: no electrical
activity
The heart has lost its electrical activity. There is no electrical pacemaker to initiate electrical flow.
230 Chapter 8
48. If the EKG only has ventricular complexes at a rate between 20 and 40 bpm, with
no P waves, you would call the arrhythmia Rhythm.
49. An EKG that is totally chaotic, with no discernible waves or complexes, and nothing
but a lot of irregular undulations, would fit the rules of .
50. Ventricular Tachycardia is a very rapid rhythm with wide, bizarre QRS complexes,
and no waves.
51. A single ectopic with a QRS greater than 0.12 second, with a T wave in the opposite
direction of the R wave and having no P wave preceding it, would be called a
Premature Complex.
52. You now have all the information you need to approach ventricular rhythms. Turn
to the Practice Strips at the end of this chapter and apply your new knowledge until
you feel very comfortable in this area.
Pulseless Electrical Activity (PEA)
53. There is a condition in which the EKG shows electrical activity that should
produce a pulse, but no pulse can be detected in the patient. This is not a rhythm itself;
it is a condition called Pulseless Electrical Activity. PEA can be seen in many rhythms,
including NSR, tachycardias, and bradycardias. PEA occurs when a heart rhythm on
the EKG does not produce the expected pulse. The key to a quick diagnosis is to take
the patient’s to see if the electrical activity is generating a
mechanical response.
Idioventricular
Ventricular Fibrillation
P
Ventricular
Practice Strips
pulse
Figure 80 Rules for Asystole
Regularity:
Rate:
P Wave:
PRI:
QRS:
straight line indicates absence of electrical activity
Ventricular Rhythms 231
54. PEA cannot be detected by looking at the EKG alone. It is necessary to take the
patient’s before the diagnosis can be made.
55. PEA often has a treatable underlying cause. One of the most common causes, and
most treatable, is hypovolemia. In order to diagnose PEA, it is necessary to look at
the while checking the patient for a corresponding
.
56. If no cause for PEA can be found, it should be treated as Asystole.
pulse
EKG
pulse
232
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